The PSA Trap: How This “Profit-Driven Health Disaster” Is Stealing Your Manhood

Let me tell you about a man I'll call Frank. He is 72 years old and a retired engineer.

His mind is sharp. He’s in shape and still plays tennis three mornings a week. He takes his wife out to dinner every Saturday night.

Then his doctor orders a “routine” PSA test.

The number comes back slightly elevated. Not dramatically — but just enough for his doctor to tell him they need to take a closer look.

Eight weeks later, Frank is recovering from a radical prostatectomy. Today, two years later, he lives with consequences he never expected.

He wears an incontinence pad. He no longer plays tennis. And he says his relationship with his wife has never been the same since the surgery.

But what troubles him most is what he learned afterward.

The cancer that was removed was classified as low-grade Gleason 6 prostate cancer — a type many experts consider slow-growing, non-aggressive, and the least likely to spread.

Many prostate researchers hesitate to even call it “cancer.”^1,2

Research has suggested that some men diagnosed with these low-risk prostate cancers may never experience symptoms or serious health problems from them during their lifetime.

Frank didn’t beat cancer. He was ambushed by a test.

The researcher who discovered PSA — Dr. Richard Ablin — spent his career arguing against its use for routine screening.

He called it “a profit-driven public health disaster.”³

He wrote a book — The Great Prostate Hoax: How Big Medicine Hijacked the PSA Test and Caused a Public Health Disaster — exposing how the urology industry and pharmaceutical companies hijacked his discovery and turned it into a billion-dollar surgery pipeline.

Why won’t your doctor listen? Because the assembly line is too profitable to shut down.

In this issue of Confidential Cures, I’ll show you why the PSA test is one of the most

dangerous medical frauds facing men today — and the five natural steps you can take to protect your prostate without ever setting foot in a surgeon’s office.

The Exposed Cover-Up:
How The Medical Cartel Turned A Flawed Blood Test
Into A Surgery Assembly Line

A landmark 2026 analysis of the CAP trial — one of the largest prostate-screening studies ever conducted — tracked 189,386 men over 15 years.

Researchers at Queen Mary University in London asked one question: How many cancers found by PSA screening are overdiagnosed — meaning they would never have caused symptoms or death?⁴

The answer should alarm every man over 65. At age 50, overdiagnosis accounts for 16%

of screen-detected cancers. Concerning, but manageable.

At age 70 — the age of many men reading this right now — it jumps to 32%. One in three.

At age 80, it hits 58%. More than half.

If you’re in your 80s and a PSA test finds “cancer,” the odds are better than a coin flip that it would never have harmed you.5

Why does overdiagnosis explode with age? It comes down to the simple reality that older men face many other potential causes of death — heart disease, stroke, or pneumonia, to name just a few.

A slow-growing Gleason 6 tumor might take 15 or 20 years to become dangerous. You’re far more likely to die with that cancer than from it.

Think of it this way: The cancer’s clock ticks slowly. Your body’s clock ticks at a normal pace. The PSA test can’t tell the difference between a tiger and a house cat. But the surgeon treats them both the same.

And the numbers bear this out. The ProtecT trial — published in the New England Journal of Medicine — followed 1,643 men with localized prostate cancer for 15 years.

They were randomized to surgery, radiation, or active monitoring.⁶

Let me be direct: There was no significant difference in prostate cancer mortality between the three groups. Men who chose to watch and wait had the same survival rate as men who went under the knife. And 39% of the monitoring group never needed any treatment at all.

What does active monitoring actually look like?

It means regular PSA checks, periodic MRI scans, and occasional biopsies to track whether anything is changing. No surgery. No radiation. No incontinence pads. Just careful, informed watching — with treatment ready if it’s ever truly needed.

For the majority of men with low-grade disease, that day never comes.

But the treatment the industry pushes is brutal.

But here’s what your surgeon won’t put in the brochure...

Research shows that nearly 7 in 10 men do not recover their sexual function to pre-surgery levels within the first year after radical prostatectomy — and for many, it never returns.¹⁰

I’m not talking about reduced function nor occasional difficulty. I mean never again.

And there’s something else. Surgery can permanently reduce penile length — in some men by up to an inch.¹¹

I’ve sat with men who weren’t told any of this before they signed the consent form. By the time they found out, it was too late.

And who profits from all of this? For an uninsured patient, a prostatectomy can cost up to $135,000.¹² And if you think having insurance protects you — that’s exactly what they’re counting on.

For cancers that, in many cases, would have stayed quiet for the rest of their natural lives.

Here’s the number that exposes the whole racket: Men have a 16% lifetime chance of being diagnosed with prostate cancer — but only a 3% chance of dying from it.¹³

That 13% gap? That’s the overdiagnosis zone. That’s where the medical cartel makes its fortune.

Even the U.S. Preventive Services Task Force — the government’s own independent panel — gives PSA screening in men 70 and older a Grade D recommendation:

Do not screen. The harm outweighs the benefits.¹⁴

Your doctor’s own professional guidelines say stop. Yet every year, millions of men over 70 are still funneled through the PSA pipeline.

I’ve been telling my patients this for over two decades. The PSA test doesn’t measure cancer. It measures inflammation — and that’s a crucial distinction.

A high PSA reading can result from something as ordinary as an infection, an enlarged prostate, recent exercise, or even sexual activity in the days before your blood draw.

None of those things are cancer. But the urology industry doesn’t profit from reassurance. It profits from biopsies. From surgeries. From radiation.

From follow-up appointments stretching out for years.

Here’s How To Protect Your Prostate And Your Independence
— Starting Today

You don’t need to choose between doing nothing and submitting to the surgical assembly line. There’s a third path — one that puts you in control.

Here are five steps for prostate health that don’t require a surgeon, a hospital gown, or surrendering your dignity:

1.     Demand Active Surveillance If Diagnosed With Low-Grade Cancer. Don’t let a Gleason 6 diagnosis — the kind doctors consider slow-growing and least likely to spread — stampede you into surgery.

As the ProtecT trial showed, active monitoring delivers the same survival outcomes as radical surgery, without the devastating side effects.¹⁵

Tell your doctor: “I want to watch this, not cut it.”

Active surveillance means regular PSA checks and periodic imaging — not abandonment. Slow-growing tumors in older men rarely progress to life-threatening disease within a natural lifespan. Monitoring catches the rare exceptions while sparing the majority from unnecessary harm.

2.      Add Beta-Sitosterol And Pumpkin Seed Oil To Your Daily Routine. This powerful plant sterol is one of nature’s best-studied answers to prostate swelling.

It works by blocking 5-alpha-reductase — the enzyme that converts testosterone into

dihydrotestosterone, or DHT, the hormone most directly linked to prostate enlargement.

A review of clinical trials found that beta-sitosterol cuts urinary symptom scores by nearly 5 points and measurably strengthens urine flow — compared to men who took a sugar pill.¹⁶

Pumpkin seed oil works alongside beta-sitosterol to reinforce the same protective pathways. It’s rich in phytosterols and has been used for centuries in European herbal medicine to support urinary flow and prostate health.

You’ll find beta-sitosterol naturally in avocados, pumpkin seeds, and pecans — but not in amounts large enough to move the needle therapeutically. Supplement with 300 mg beta-sitosterol daily and 2 tablespoons of pumpkin seed oil.

Choose a beta-sitosterol formula standardized to genuine beta-sitosterol, not a generic plant sterol blend.

3.     Load Up On Lycopene With Cooked Tomatoes And Watermelon. The compound you’re after is lycopene — one of nature’s most concentrated antioxidants, and one that accumulates specifically in prostate tissue.

A meta-analysis of 42 studies covering 692,012 participants found that higher lycopene intake is associated with a 12% reduction in prostate cancer risk.¹⁷

Here’s a key fact you might not know: Cooking dramatically increases lycopene’s bioavailability.

Raw tomatoes deliver some lycopene, but marinara sauce, tomato paste, and stewed tomatoes deliver far more because heat breaks down the cell walls and releases the compound.

Watermelon is one of the few raw sources that competes. Aim for 10 to 15 mg of lycopene daily

— roughly two tablespoons of tomato paste or two cups of watermelon. Supplement with 10 mg daily.

4.     Protect Your Prostate With Selenium And Zinc. These two minerals are essential to prostate cell defense, and both become depleted with age.

A Harvard study found that men who got the most selenium had the healthiest prostates. This was true even for men with high PSA levels. If they were in the group with the most selenium, their prostate cancer risk dropped by 50%.¹⁸

Another study, titled The Netherlands Cohort Study, followed almost 60,000 men. It found those with the highest selenium had the lowest risk for prostate cancer.¹⁹

Zinc is even more critical — the glandular cells of the human prostate accumulate more zinc than any other soft tissue in the body, and that high concentration is directly linked to normal prostate function.²⁰ When zinc levels fall, the prostate loses a key line of defense.

One important note: taking supplemental zinc over time can deplete copper, another mineral your body needs for cellular repair and immune function. To keep these minerals in balance, add a small amount of copper alongside your zinc.

For selenium, the best food sources are Brazil nuts — just two or three a day provide close to your daily target. Oysters, sunflower seeds, and sardines are also good sources. For zinc, reach for oysters, grass-fed beef, pumpkin seeds, and cashews.

Supplement with 200 mcg selenium in an organic form your body can absorb easily — look for selenomethionine or selenocysteine on the label. Take 30 mg zinc picolinate daily — the most bioavailable form — along with 900 mcg copper to maintain the proper balance between the two minerals.

5.     Eat Cruciferous Vegetables Daily. Cauliflower, Brussels sprouts, and broccoli, contain two compounds — sulforaphane and indole-3-carbinol — that research shows can directly block the growth of prostate cancer cells.²¹

They work by supporting healthy hormone metabolism — specifically by helping your body clear excess estrogen through cleaner pathways, which matters for prostate health more than most men realize.

A half-cup of broccoli or Brussels sprouts at dinner covers your daily cruciferous requirement.

You can supplement with 100 mg of indole-3-carbinol daily or 20 mg of sulforaphane.

These five steps won’t make headlines in a medical journal — but they’re what I recommend to the men who come to my clinic, and they work.

The medical cartel wants you afraid. Afraid means compliant. And compliance means more profit.

But now you have what they never wanted you to see: the data. The discoverer’s own warning. The proof that watching and waiting is just as safe as the knife. And a natural plan that supports your prostate every single day.

 
To Your Good Health,

Al Sears, MD, CNS

1. Worthington F. “Gleason 6 Prostate Cancer: An Expert Pathologist’s View.” www. pcf.org/gleason-336-is-not-not-cancer/

2. Burke H. “Gleason 6 prostate cancer: That which cannot be named.” Front Oncol. 2022 Dec 5;12:1073580.

3. Ablin RJ and Piana R. The Great Prostate Hoax: How Big Medicine Hijacked the PSA Test and Caused a Public Health Disaster. Palgrave Macmillan. 2014.

4. Brentnall AR, et al. “Evaluating the impact of age on prostate cancer overdiagnosis using long-term follow-up from the CAP randomised trial.” International Journal of Cancer. 2026; DOI: 10.1002/ijc.70492.

5. Brentnall AR, et al. “Evaluating the impact of age on prostate cancer overdiagnosis using long-term follow-up from the CAP randomised trial.” International Journal of Cancer. 2026; DOI: 10.1002/ijc.70492.

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7. Haglind E, et al. “Urinary incontinence and erectile dysfunction after robotic versus open radical prostatectomy: a prospective, controlled, nonrandomised trial.” Eur Urol. 2015;68(2):216–225.

8. Incrocci L. “Radiotherapy for prostate cancer and sexual health.” Transl Androl Urol. 2015;4(2):124–130.

9. Johns Hopkins Medicine. “Bowel dysfunction after prostate cancer treatment.” hopkinsmedicine.org. Updated February 2024.

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13.    Brawley OW. “Trends in prostate cancer in the United States.” J Natl Cancer Inst Monogr. 2012;2012(45):152–156.

14.    U.S. Preventive Services Task Force. “Screening for prostate cancer: US Preventive Services Task Force recommendation statement.” JAMA. 2018;319(18):1901–1913.

15.    Hamdy FC, et al. “Fifteen-year outcomes after monitoring, surgery, or radiotherapy for prostate cancer.” N Engl J Med. 2023;388(17):1547–1558.

16.   Wilt T, et al. “Beta-sitosterols for benign prostatic hyperplasia.” Cochrane Database Syst Rev. 2000;(2):CD001043.

17.    Rowles JL, et al. “Increased dietary and circulating lycopene are associated with reduced prostate cancer risk: a systematic review and meta-analysis.” Prostate Cancer Prostatic Dis. 2017;20(4):361–377.

18.    Li H, et al. “A prospective study of plasma selenium levels and prostate cancer risk.”
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19.    Geybels M, et al. “Advanced prostate cancer risk in relation to toenail selenium levels.” J Natl Cancer Inst. 2013 Sep 18;105(18):1394-401.

20.    Costello LC, et al. “Evidence for a zinc uptake transporter in human prostate cancer cells which is regulated by prolactin and testosterone.” J Biol Chem. 1999;274(25):17499–504.

21.    Frydoonfar HR, et al. “The effect of indole-3-carbinol and sulforaphane on a prostate cancer cell line.” ANZ J Surg. 2003;73(3):154–156.